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Since reduction of IOP in some patients fails to blunt the damage to OHN and/or reduce the loss of RGCs and the patients continue to lose visual acuity, it has been recognized that an important goal for treating glaucoma is to reduce or attenuate the progression of RGC death in addition to reducing ocular hypertension. The etiology of optic neuropathy has many routes spanning many different mechanisms that result in RGC demise and optic nerve damage, including mechanical constriction and vasoconstriction of retinal blood vessels, and increased sensitivity of RGCs

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