Obsessive compulsive disorder

Neurocircuitry models of OCD suggest that OCD symptoms are mediated by hyperactivity in orbitofrontal-subcortical circuits, with a primary pathology centered in the striatum.15 Functional imaging studies have demonstrated increased activity in orbitofrontal and anterior cingulate cortices in neutral state paradigms, which is accentuated in symptom provocation and cognitive activation paradigms, and attenuates with successful treatment.19 Increased metabolism in the striatum has also been observed in untreated OCD patients and correlates well with the degree of symptom severity.

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