In the real world, it is likely that not only one of the scenarios depicted, but a combination of several ones, may affect how well a patient responds to a given treatment, or how likely it is that he or she will suffer an adverse event. Thus, a fast-metabolizing patient with poor-responder pharmacodynamics may be particularly unlikely to gain any benefit from taking the drug in question, while a slow-metabolizing status may counterbalance in another patient the same inopportune pharmacodynamics, while a third patient, who is a slow metabolizer and displaying normal pharmaco-dynamics, may be more likely to suffer adverse events. In all of them, both the pharmacokinetic and pharmacodynamics properties may result from the interaction of several of the mechanisms described above. In addition, we know of course that coadministration of other drugs or even the consumption of certain foods may affect and further complicate the picture for any given treatment.
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