Cure Eye Floaters Naturally

Eye Floaters No More

Eye Floaters (Also known as Eye Flashes) are deposits of various sizes and shapes that float within the eye. They are caused by degenerative changes of the vitreous humour the clear gel that fills the eyeball. Eye infections, inflammation, wounds and damage to the eye can lead to eye floaters. A sudden increase in floaters can be one of the first signs of retinal detachment or other severe eye conditions. In Eye Floaters No More, you'll discover: How to finally get rid of your stressful eye floaters, blocks of vision, the flashing lights using a safe, natural and easy system. Eliminate your annoying eye floaters from the comfort of your home. How to prevent more eye floaters from forming. How to find out if your eye floaters are a sign of other eye conditions. Easy, natural ways to drastically improve your vision. More here...

Eye Floaters No More Overview


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Age Related Macular Degeneration

When considering potential causes and treatments of macular degeneration, it is informative to consider the normal vision process from a functional and morphological standpoint. Light that is transmitted through the surface and the anterior chamber of the eye is absorbed in the outer retina by opsin-bound (as a Schiff base) retinaldehyde, which is present in specialized light-gathering cells called photoreceptors. There are two types of photoreceptors rods, which are used primarily for low light vision, and cones, which are responsible for color perception and visual acuity. Light-induced isomerization of the retinaldehyde Schiff base position 11 olefin from cis to trans geometry generates a signal, which is converted to a nerve impulse for eventual relay to RGCs in the inner retina (Figure 2). RGCs transmit this signal through the optic nerve head (ONH) and into the brain, where the signal is integrated.

Macular Degeneration

The part of the retina (the light-sensitive membrane that lines the back of the eye) that provides sharp sight in the center of the field of vision is called the macula. You need this sharp central vision to see fine details. It is essential for driving, reading, and recognizing faces. As you age, the tissue of the macula can become damaged, leading to a loss of central vision. Age-related macular degeneration is the leading cause of blindness in the United States. There are two general types of macular degeneration dry and wet. The dry form accounts for about 90 percent of all cases of the disorder. In this form, the macula slowly thins until vision becomes dimmed. Although only about 10 percent of all people with macular degeneration have the wet form, they are at much higher risk of losing their sharp central vision than are those with the dry form. New blood vessels grow under the retina in the wet form of the disease. These The greatest risk factor for macular degeneration is...

Evaluation of Eyelid Malpositions

Snapback Test

A history of previous eye surgery is important. Eyelid malpositions are not uncommon sequelae of retinal detachment surgery, strabismus surgery or cataract extraction. Ptosis is reported to occur following cataract surgery in 7-8 of cases. Other surgery, especially intracranial or thoracic procedures, may result in central third nerve palsy or Horner's syndrome respectively.

The electrically stimulated retina

Photoreceptor loss due to retinal degenerative diseases such as age-related macular degeneration and retinitis pigmentosa is a leading cause of blindness. Although these patients are blind, they possess functioning bipolar and ganglion cells that relay retinal input to the brain. Retina implants are in development in order to evoke optical sensations in these patients. The success of such an approach to provide useful vision depends on elucidating the neuronal target of stimulation. A compartmental model for extracellular electrical stimulation of the retinal ganglion cell (RGC) has been developed.71 In this model, a RGC is stimulated by extracellular electrical fields with active channels and realistic cell morphology derived directly from neuronal tracing of an amphibian retina cell.

Primary Nursing Diagnosis

Complications may include retinal disorders, pupillary block, adhesions, acute glaucoma, macular edema, and retinal detachment. Following extracapsular cataract extraction, the posterior capsule may become opacified. This condition, called a secondary membrane or after-cataract, occurs when subcapsular lens epithelial cells regenerate lens fibers, which obstruct vision. After-cataract is treated by yttrium-aluminum-garnet (YAG) laser treatment to the affected tissue.

Introduction Ocular Anatomy and Diseases

Most information about our surroundings is gathered by the eye, which is literally a 'window' for the brain. With an aging population, the prevalence of sight-threatening ocular diseases continues to increase. Thus, for instance, more than 70 million people suffer from glaucoma worldwide.1-3 Visual impairment caused by diabetes affects up to 90 of diabetics over 10 years of age. Likewise, pathological dry eye and ocular allergic conditions afflict > 100 million patients worldwide, and age-related macular degeneration (AMD) is the leading cause of blindness among the elderly, affecting up to 28 of patients after the seventh decade of life.2 Consequently, the discovery and development of therapeutic products for the treatment of these various ocular diseases is of paramount importance, and is being actively pursued within the pharmaceutical industry.1-3

Figure 7 The structures of 5fluoruracil and mitomycin C

The disadvantages to pegaptanib therapy arise from drug specific and drug class considerations. Pegaptanib is administered by intravitreal injection, which is inconvenient, requires highly skilled delivery by an ophthalmologist, and in clinical trials has demonstrated a higher rate of intraocular infection (endophthalmitis) and retinal detachment than in controls.67 It is not known if pegaptanib's lack of binding to other VEGF isoforms lessens its effectiveness as compared with pan-isoform binders like the anti-VEGFantibody ranibizumab (see Section Pegaptanib does not improve visual acuity or arrest its degradation, which would be ultimately desirable.

Stat5a 5b Crystallization

Gcsf Bcl2 Caspase

Retinal hypoxia and excessive secretion of VEGF can lead to an inappropriate retinal vascularization126 and hemorrhages contributing to visual loss observed in retinopathy of prematurity, diabetic retinopathy, as well as age-related macular degeneration.127 Antiangiogenic therapies are now under investigation in order to prevent such retinal neovascularization. In the case of wet age-related macular degeneration, such treatment consists of lucentis, an antibody fragment designed to bind all forms of VEGF (ranibizumab, rhuFabV2, Genentech) and in the angiostatic steroid, Retaane (anecortave acetate, Alcon Laboratories),128 which are both in phase III of clinical trials,129 whereas Macugen (pegaptanib sodium injection, Eyetech Pharmaceuticals), an aptamer specific for the VEGF165 isoform, has recently received US FDA approval.130 Various others drugs for the treatment of age-related macular degeneration, such as AdPEDF (Genvec),131 squalamine (Genaera),132 and combrestatin A4 prodrug...

Pathologies leading to blindness

The leading causes of untreatable blindness are age-related macular degeneration (AMD), retinitis pigmentosa (RP), accidents, and cancers. Additionally, Age-related macular degeneration is pathology of the retina characterized by the progressive loss of central vision, generally occurring in the elderly. It is the leading cause of blindness in developed countries, with an estimated 2,000,000 individuals having some form of AMD in the United States. For unknown reasons, the pigmented epithelium of the retina degenerates, leading to a subsequent degeneration of the photoreceptors and fluid leakage into the neural retina. As this occurs in the macular region, central vision is compromised, leading to the loss of the ability to distinguish fine detail. Retinitis pigmentosa is an inherited pathology of the retina characterized by the loss of peripheral vision and night vision. An estimated 100,000 individuals are affected by RP in the United States. For unknown reasons, the rod...

Optic nerve neuroprosthetic approach

The optic nerves provide the sole conduits of visual information from the retina to the lateral geniculate nucleus in the thalamus. In certain visual pathologies, such as age related macular degeneration or retinitis pigmentosa, where a subpopulation of ganglion cells has been spared, but in which cells of the outer retina are completely degenerate, the optic nerve offers a possible site for intervention via a neural interface.

As Sources Of Distress

The advent of HAART therapy has altered the natural progression of HIV and has changed the incidence, natural history, management, and sequelae of HIV-associated retinopathy, especially CMV-associated retinopathy. Before use of HAART, CMV retinitis was common, occurring in 20 -40 of seropositive patients. Patients were relegated to indefinite intravenous therapy, and between 25 and 50 suffered retinal detachment. Survival after development of CMV retinitis was 6-10 months. The incidence of CMV retinitis declined by approximately 80 after the advent of HAART therapy, and mean survival has increased to over 1 year from time ofdiagnosis (Holbrook et al., 2003 Goldberg et al., 2005). However, visual loss and blindness from multiple etiologies are still significant causes for concern and sources of distress for patients with HIV (Ng et al., 2000 Kestelyn and Cunningham 2001 Hill and Dubey 2002 Oette et al., 2005). Specific studies examining the quality of life and distress experienced by...

Retinal anatomy and physiology

Retinal Anatomy

Figure 11.2 Normal human retina as viewed through an ophthalmoscope, highlighting the regional specialization of the retina. The optic nerve head, also known as the optic disk, is where the axons of the retinal ganglion cells are collected together into the optic nerve as these axons course towards their subcortical targets. This region is devoid of photoreceptors. Additionally, the blood vessel of the retinal circulation originates from the optic nerve head. The fovea, the region of darker pigmentation, subserves the central 6 degrees of visual space and is the region of greatest visual acuity. The photoreceptors in this region are primarily cones. The macula lutea, outlined with a dashed line, subserves the central 20 degrees of visual space. Both rod and cone photoreceptors are found outside the fovea, with rod photoreceptors becoming more prevalent the farther one is from the fovea. The two principal sources of untreatable blindness, age-related macular degeneration and retinitis...

Surgical Retinal Procedures

K retinal detachment occurs when the retina is pulled away from or out of its normal position. Approximately 5 of the U.S. population has retinal breaks, but most do not lead to retinal detachment, which has a prevalence of 0.3 . Estimates are that 15 of people with retinal detachments in one eye develop detachment in the other eye, and the risk of bilateral detachment increases to 30 in people who have had bilateral cataract surgery. 816 Retinal Detachment optic nerve and at the ciliary body. The remaining retina relies on the vitreous (jelly-like mass that fills the cavity of the eyeball) to apply pressure against the lining to maintain its position. The detachment can occur spontaneously as a result of a change in the retina or vitreous this detachment is referred to as a primary detachment. Secondary detachment occurs as a result of another problem, such as trauma, diabetes, or pregnancy-induced hypertension. Complications from retinal detachment include visual impairment and...

Figure 2 11cis and 11fransretinaldehyde

A likely functionally important component of lipofuscin is the amphiphilic pyridinium ion A2E19 (Figure 3), that is believed to be formed in vivo from the condensation of one equivalent of phosphatidylethanolamine with two equivalents of 11-trans-retinaldehyde, followed by phospholipase-D-catalyzed dephosphorylation. A2E might be the major component of lipofuscin, producing reactive oxygen species (ROS).20 A2E is thought to be the major component of lipofucsin producing ROS in the presence of light and oxygen.20 Enhanced production of A2E can occur as the result of the dysfunctional transport of photobleached 11-trans retinal phosphatidyethanolamine Schiff base (NRPE) through the photoreceptor disk membrane resulting in the accumulation of RPE cell lysosomes. BNRPE transport ossurs via an ABCR (ATP-binding cassette transporter) mechanism. Individuals null for the ABCR gene suffer from Stardgardt's macular dystrophy, an early-onset macular degeneration-like disease.21 Individuals with...

Other Diseases of the Retina

Diabetic retinopathy (DR) occurs in 27 of diabetics after 5-10 years of contracting diabetes and up to 90 have DR after more than 10 years of diabetes. DR essentially stems from retinal ischemia due to thickening of retinal capillary basement membranes when capillary pericytes start to die off. The ensuing ischemia causes the overexpression of VEGF, which in turn causes capillaries to leak and cause edema. VEGF also stimulates the growth of new blood vessels. This neovascularization could be perceived as a compensatory mechanism, but it is actually detrimental, since the new blood vessels disrupt retinal function. Deterioration of vision in DR is due to macular edema and proliferation of fibrovascular membranes that can lead to retinal detachment. Current treatments for DR are limited to laser photocoagulation of the leaky blood vessels, but this unfortunately destroys the underlying and surrounding retinal tissue. A much more specific approach is the use of photodynamic therapy,...

Ophthalmic agents

The eye is a key part of the CNS connecting to the brain via the second cranial or optic nerve. Many other receptors and signal transduction process in the eye recapitulate those present in the CNS proper although the cellular architecture of the eye and its function are unique (see 6.12 Ophthalmic Agents). The anterior pole of the eye, composed of the cornea, iris, and lens, serves to focus light onto the photoreceptors of the retina. The retina is a layered structure composed of retinal ganglion cells, amacrine cells, horizontal cells, and photoreceptors that transduces, processes, and integrates visual stimuli. The ganglion cells are the output neurons of the retina, sending axons to the lateral geniculate nucleus by way of the optic nerve. This highly evolved complex system allows for accurate processing of visual stimuli with an exceptional dynamic range. However, it is susceptible to multiple disorders including glaucomas and macular degeneration that ultimately can produce...

Patient Interview

Occurred during a lecture and produced a 'thick mist' in front of his eye within 15 minutes, an operation was necessary. This was not a success and Marcus became blind in his right eye. The other eye was also suffering. Despite laser treatment and removal of the vitreous humour, the doctors at a specialist clinic could not save it and Marcus now has only 3 vision in this eye. 'That's enough, in good light, to see someone opposite me as a dark shape.'


There is a strong precedence for a DNA-based oligonucleotide drug as a therapeutic for cytomegalovirus induced retinitis (Vitravene) and the registration of the first siRNA drug is only a question of time. The first clinical trial for a siRNA-based therapeutics has been already initiated. Sirna-27 is a chemically modified siRNA that targets the mRNA of VEGFR-1. This siRNA is used to inhibit ocular neovascularization associated with age-related macular degeneration and the compound is administered by intraocular injection. A more difficult question to answer at present is whether applications will go beyond these niches.

Retinal Degeneration

Retinal degeneration is a well-studied system where genes producing apo-ptotic cell death and photoreceptor loss have been analyzed in animal models and in human disease.36-41 The involved genes result in photoreceptor loss with resultant retinitis pigmentosa in early to midlife with devastating effects and loss of vision. Other pro- and antiapoptotic and related genes are activated during this process.42-45 The directly involved defective genes, such as rhodopsin, are involved in signal transduction. The defective proteins result apparently in altered G-protein-related signaling so as to promote a balance favoring apoptotic cell death. This imbalance, since it involves signaling mechanisms for physiological transduction of vision, can also be influenced by environmental factors such as light flux, ischemia, or retinal detachment.46-48 Thus, in experimental models, the process of apoptotic cell death can be triggered by altering the light and wavelength input to the retinal tissue at...

Optobionics group

Additionally, the implants are reported to have improved the recipient's visual function however, these results are only available as recipient testimonials, not controlled scientific studies. Care must be taken to distinguish the improvement due to the activity of the device and the improvement that occurs simply by the introduction of the device. It has been established that a transient, functional rescue of a dysfunctional retina occurs in animal models as the result of surgical intervention into the subretinal space, even if the intervention does nothing more than expose the subretinal space. Nevertheless, the test of the biocompati-bility of the materials and the development of the implant techniques make the ASR a groundbreaking device.


CLINICAL PRESENTATION Orbital involvement is seen in 80 of patients with mucormycosis, and 11 will progress to cavernous sinus thrombosis. Patients usually present with impaired ocular movement, loss of vision, proptosis, chemosis, and periorbital cellulitis. An orbital apex syndrome with blindness and total ophthalmoplegia may be seen. Serous retinal detachment may result from inflammation of the sclera. Rare cases of fungal enophthalmitis result from angioinvasion by fungal hyphae. With eyelid involvement cutaneous lesions may appear as large necrotic and ulcerating lesions with erythematous borders, and oozing black pus. Sinusitis and nasal discharge occur and nasal exam reveals a thick, dark blood-tinged discharge and reddish black necrotic eschar on the turbinates and septum. Cerebral involvement and hemiparesis can be seen in 15 to 20 of cases. Major systemic signs and symptoms include lethargy and headache.

Operating Principle

The OFM imaging method can be best understood by taking a closer look at the first two aforementioned functions of a conventional microscope image replication and magnification. We can do away with the need for image replication if we are willing to rethink the way we use a CCD or CMOS sensor grid. Unlike the human retina, we can place the object of interest directly onto such a sensor grid. By illuminating the object uniformly, an image of the object can be directly recorded by the sensor grid. The absence of optical elements in this arrangement implies that there is no aberration or collection numerical aperture to worry about. This imaging strategy mirrors the phenomenon of floaters in our eyes and has been implemented as an imaging method recently 11, 12 .

Nevus Flammeus

Choroidal Capillary Hemangioma

CLINICAL PRESENTATION Nevus flammeus presents as a flat purple or deep red vascular lesion that can vary from only a few millimeters in size to those covering vary large areas. On the face it is usually unilateral and in the distribution of one or more branches of the trigeminal nerve. Unlike other congenital eyelid vascular lesions it does not undergo spontaneous regression. In older adults nevus flammeus can undergo cavernous changes making them elevated and rather prominent. Pyogenic granuloma may arise within a nevus flammeus without any predisposing factors, as can rare cases of basal cell carcinoma. In the presence of Sturge-Weber syndrome associated ocular manifestations include diffuse choroidal hemangioma, ipsilateral glaucoma, and serous retinal detachment. Cases of acquired nevus flammeus have been described following trauma.

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