Learning Disabilities Ebooks Catalog

Adult Dyslexia

Adult Dyslexia

This is a comprehensive guide covering the basics of dyslexia to a wide range of diagnostic procedures and tips to help you manage with your symptoms. These tips and tricks have been used on people with dyslexia of every varying degree and with great success. People just like yourself that suffer with adult dyslexia now feel more comfortable and relaxed in social and work situations.

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Dyslexia Facts You Should Know

Dyslexia, Facts You Should Know Is A Unique Fact Filled E-book That Gathers Easy To Read Information About Dyslexia In One Place For The Interested Reader. Learn How to Cope With A Diagnosis of Dyslexia and What To Do Next. Every bit of this eBook is packed with the latest cutting edge information on Dyslexia. It took months to research, edit, and compile it into this intriguing new eBook. Here's what you'll discover in Dyslexia: Facts You Should Know: What is Dyslexia? History of Dyslexia. Is it Dyslexia or Something Else? How You Can Diagnose Yourself. Benefits of Catching Dyslexia Early. What is the Dyslexia Test? How to Get Everything You Will Need for Help in Coping with Dyslexia. Exploring Your Options for Schools and Programs. What is the Individualized Education Program? Alternatives to the Iep. The Roll Your Childs Teacher Plays. Your Part in Your Childs Education. Why Practice, Patience and Practicality are Most Important. Teens with Dyslexia. Success in Life: Adults Overcoming Dyslexia.

Dyslexia Facts You Should Know Summary


4.6 stars out of 11 votes

Contents: Ebook
Author: Dee Henry
Price: $27.00

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Developmental Learning Disability

About three decades ago, I was listening to Norman Geschwind speak about dyslexia to members of the Orton (dyslexia) Society. Norman liked to say things that would shock his audience, and during his talk he said, Dyslexia is a social disease. At that time when someone spoke about social diseases, they were usually speaking about sexually transmitted diseases, such as gonorrhea and syphilis. There were many dyslexic people and parents of dyslexic children in the audience who appeared to be angry when he made this comment. He, however, held up his hand and said, Wait, let me explain. He mentioned that in the history of humans, it is only recently that reading has become so critical for success. He asked who would be our leader if we lived in a hunter-gatherer society the person who could read and write the best or the person who could get us back to camp after the hunt He also mentioned that he would not be surprised if the people with the best visual-spatial abilities have a higher...

Manifestations of the Spectrum of Mps I Disease

The broad range of severity, both in overall disease status as well as in individual components of the disease, leads to a very heterogeneous population of patients in varying stages of disease progression within the various body systems. At the severe end of the disease spectrum, Hurler patients have severe developmental problems, whereas Hurler-Scheie patients show little or no mental retardation but may experience learning disabilities. Scheie patients do not lose intellectual function, in general. Hurler patients can experience cardiac failure due to stiffening of the heart as well as coronary artery disease. Their valvular disease does not progress fast enough in most cases to be the primary cardiac problem. In contrast, intermediate and milder patients have valvular disease problem as their dominant cardiac problem, often beginning at age 8 to 10, and continuing through their teenage years and

Preparing information for the seminar

Everyone enjoys preparing slides in PowerPoint because of the animated features and sound effects. These should be used with some caution in presentations as they can become a major distraction and eventually an irritation to your audience. Anyone who has dyslexia will also find your presentation very difficult to follow, particularly where blocks of text are animated on a slide.

Central Auditory Development in Children with Cochlear Implants Clinical Implications

Abr Latencies Infants

A common finding in developmental neurobiology is that stimulation must be delivered to a sensory system within a narrow window of time (a sensitive period) during development in order for that sensory system to develop normally. Experiments with congenitally deaf children have allowed us to establish the existence and time limits of a sensitive period for the development of central auditory pathways in humans. Using the latency of cortical auditory evoked potentials (CAEPs) as a measure we have found that central auditory pathways are maximally plastic for a period of about 3.5 years. If the stimulation is delivered within that period CAEP latencies reach age-normal values within 3-6 months after stimulation. However, if stimulation is withheld for more than 7 years, CAEP latencies decrease significantly over a period of approximately 1 month following the onset of stimulation. They then remain constant or change very slowly over months or years. The lack of development of the...

Implants in Children with Multiple Disabilities andor Auditory Neuropathy

Our ability to implant children with a cochlear device has no more serious potential for damage and financial waste than in instances in which the child is deaf but has additional disabilities. It must be admitted, if not generally acknowledged, that children with multiple disabilities, including mental retardation or severe language-learning difficulties, have been implanted already. In some children who are deaf and blind but otherwise intact, the decision to implant may be a good one. However, the parents of children with multiple disabilities, including deafness, are among the most vulnerable as they seek help for the child whose future may seem bleak. Everyone must understand the implant's role as only one factor in the habilitation and

Brain Size

As I mentioned earlier, Marion Diamond et al. (1985) found a higher glial cell-to-neuron ratio in Einstein's brain than they did in control individuals. Glial cells perform many functions, but their primary functions are structural and supportive. The axons that travel below the cortex and connect anatomically separate areas of cortex have a coating on them that is called myelin, and it is this coating that gives the subcortical white matter its characteristic color. Glial cells called oligodentrocites produce myelin. Although Einstein's high glial ratio might have been related to Einstein's dyslexia, it is also possible that this increased ratio was evidence of a high level of connectivity.

Sexspecific Behavior

Another behavioral sign of a sexually distinct nervous system comes from the prevalence of certain neurological and psychiatric diseases in males versus females. For example, both dyslexia and schizophrenia are more prominent in males (about 75 of cases), while anorexia nervosa is exhibited primarily by females (over 90 of cases). Many studies have also focused on the cognitive abilities of normal adult humans (Kimura, 1996). When presented with two figures drawn at different orientations, males are better able to mentally rotate the objects to determine whether the two figures are the same. In contrast, when presented with a picture containing many objects, females are better able to say which objects have been moved in a second picture. While these results tend to fascinate us, the challenge will be to understand what exactly is being measured and what its relevance is to behavior.

Social Adjustment

One important aspect of school adjustment pertains to the extent to which the child participates in athletics and other extracurricular activities. These activities indicate how well the person is socially integrated and accepted by peers. In addition, it is essential to evaluate academic achievement and learning aptitude in the basic skill areas. For example, learning disability compounded by low self-esteem may be a major factor propelling a youngster toward drug use, as well as other non-normative behaviors. Standardized learning and achievement tests can readily document whether a learning deficit is present.


In chapter 2 I further discuss the relationship between intelligence and creativity. As I discuss in chapter 3, many people who have been considered to be geniuses have learning disabilities. It is doubtful that these people with verbal or math learning disabilities would have scored higher than 140 in IQ testing. Two of perhaps the most creative scientists in the modern era, Einstein and Darwin, demonstrate the relationship between learning disability and creativity. Einstein had trouble with learning to read (developmental dyslexia) and in doing arithmetic (developmental dyscalculia). In his autobiography Darwin wrote that, compared with his younger sister, he was very slow to learn, and his teachers considered him as a very ordinary boy who was below the common standard in intellect. Children with developmental language disorders including dyslexia often have problems learning foreign languages. Like Einstein, Darwin might have had a developmental language disorder because he wrote...


Since the pioneering reports of Paul Broca, studies of patients with discrete brain lesions suggest that a person can have specific cognitive disabilities. Developmental disorders can also be associated with specific cognitive disabilities. For example, there are children with specific disabilities in reading, math, drawing, music, and route finding. Some of these children become creative geniuses. For example, as previously mentioned, there are many great artists, such as Picasso, who did not do well in school because of language disabilities and, as it was noted in chapter 1, even mathematical geniuses such as Einstein have had language learning disabilities. Thus, the g factor alone cannot explain specific disabilities or specific talents, and several theorists have placed more of an emphasis on special factors. For example, Howard Gardner (1985), who popularized the concept of multiple intelligences, had worked with neuropsychologists and behavioral neurologists such as Edith...