Polycystic Ovary Syndrome Natural Treatment

The Natural Pcos Diet

The Natural Pcos Diet, By Jenny Blondel, A Leading Australian Naturopath In Response To Thousands Of Requests For Professional Information To Help Women Suffering From Pcos. Real Solutions To Naturally Overcome PCOS. Naturally balance your hormones Increase your chances of conceiving Help you lose weight and feel good Curb your cravings for sugary foods Turn your fatigue around Achieve clearer, glowing skin See improvements in your mood. Do You Feel PCOS Is. Ruling Your Life? At Last! The Natural PCOS Diet. A Naturopath’s Easy Step-by-Step Guide to Overcoming PCOS Is. Now Available! Read more here...

The Natural Pcos Diet Summary

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I usually find books written on this category hard to understand and full of jargon. But the author was capable of presenting advanced techniques in an extremely easy to understand language.

This e-book served its purpose to the maximum level. I am glad that I purchased it. If you are interested in this field, this is a must have.

Polycystic Ovary Syndrome Diabetes and Cardivascular Disease

PCOS is the most common endocrinopathy that affects women of reproductive age (177). Data on the exact prevalence are variable mostly because of the lack of well-accepted diagnostic criteria. At present, the diagnosis of PCOS is based on the presence of ovulatory dysfunction and clinical or biochemical evidence of hyperandrogenism. The diagnosis requires a complete evaluation for exclusion of other causes of hyperandrogenism such as nonclassic adrenal 21-hydroxylase deficiency and androgen secreting neoplasms. The presence of polycystic ovaries on ultrasound is not a criterion for diagnosis as this is commonly found in randomly selected women (178). Although PCOS is known to be associated with reproductive morbidity and increased risk for endometrial cancer, diagnosis is especially important because PCOS is now thought to increase metabolic and cardiovascular risks (179). These risks are strongly linked to insulin resistance which is present in both obese and lean women with PCOS.

Polycystic Ovary Syndrome and Cardiovascular Risk

PCOS is associated with an increase in cardiovascular risk factors (189). In addition to obesity that is commonly present and independently associated with increased cardiovascular risk, women with PCOS have dyslipedemia, hypertension and elevated PAI-1 levels. Obesity is a prominent feature in women with PCOS as about half of the patients are obese. Also, obesity appears to confer an additive and synergistic effect on the mani Women with PCOS have higher serum triglycerides, total and LDL cholesterol and lower HDL cholesterol levels than weight-matched regularly menstruating women (190). These findings however, vary and depend on the weight, diet and ethnic background. In a large study of non-Hispanic white women, elevated LDL-C was the predominant lipid abnormality in women with PCOS (191). An additional parameter contributing to the elevated cardiovascular risk is hypertension. Obese women with PCOS have an increased incidence of hypertension and sustained hypertension is threefold...

Effect of Insulin Resistance Treatment on Polycystic Ovary Syndrome Weight Loss

Weight reduction is of paramount importance and cornerstone of every therapeutic strategy in PCOS. Although obesity does not seem to be the primary insult in PCOS, many studies have demonstrated the beneficial impact of weight reduction on the manifestations of the syndrome and especially insulin sensitivity, risk for diabetes and adverse cardiovascular risk profile (199). The effect of weight reduction by a hypocaloric low-fat diet on the metabolic and endocrine variables was studied in obese women with PCOS In another study, the effect of dietary intervention on insulin sensitivity and lipids, fibrinolysis and coagulation was examined also in obese women with PCOS (201). Insulin sensitivity was assessed by the eyglycemic clamp technique before and after a very low-calorie, protein-rich diet for 4 weeks that was followed by a low-calorie, low-fat diet for 20 weeks. After the 24-week intervention, insulin sensitivity was significantly increased along with a significant reduction of...

Pfiesteria Complex Organisms PCOs

Agents Pfiesteria piscicida and 10-12 related dinoflagellates, all collectively known as Pfies-teria-Complex Organisms (PCOs). Toxins (1) Water-soluble, ichthyotoxic neu-rotoxin (2) lipid-soluble necrotoxin causing punched out necrotic fish lesions. LD50 No human fatalities, but neurotoxin is always lethal in fish.

Step 5 Followup laboratory evaluation

A high serum androgen value may suggest the diagnosis of polycystic ovary syndrome or may suggest an androgen-secreting tumor of the ovary or adrenal gland. Further testing for a tumor might include a 24-hour urine collection for cortisol and 17-ketosteroids, determination of serum 17-hydroxyprogesterone after intravenous injection of corticotropin (ACTH), and a dexamethasone suppression test. Elevation of 17-ketosteroids, DHEA-S, or 17-hydroxyprogesterone is more consistent with an adrenal, rather than ovarian, source of excess androgen.

Diabetes and fibrinolysis

Decreased fibrinolytic system capacity is observed consistently in blood from patients with DM, particularly those with type 2 diabetes (93,94). It has been known for many years that obesity is associated with impaired fibrinolysis (95) that elevated blood triglycerides and other hallmarks of hyperinsulinemia are associated with increased activity of PAI-1 (96) and that elevated PAI-1 is a marker of increased risk of acute MI as judged from its presence in survivors compared with age-matched subjects who had not experienced any manifestations of overt CAD (97). We found that impaired fibrinolysis in subjects with type 2 DM, not only under baseline conditions but also in response to physiological challenge, was attributable to augmented concentrations in blood of circulating PAI-1. Furthermore, obese diabetic subjects exhibited threefold elevations of PAI-1 in blood compared with values in nondiabetic subjects despite tissue-type plasminogen activator (t-PA) values that were virtually...

Mechanisms responsible for the overexpression of pai1 in diabetes

Therapy designed to reduce insulin resistance, the resultant hyperinsulinemia, or both have been shown to reduce PAI-1 in blood as well. Thus, treatment of women with the polycystic ovarian syndrome with metformin or troglitazone decreased concentrations in blood of insulin and of PAI-1 (100,103). Changes in the concentrations of PAI-1 in blood correlated significantly with those of insulin (100). The concordance supports the view that insulin contributes to the increased PAI-1 expression seen in vivo. indicative of syndromes of insulin resistance such as high body mass index and waist to hip ratio in addition to advanced age and elevated concentrations of triglycerides, the association of PAI-1 activity with physical activity was no longer significant. This observation, particularly in combination with the results seen after therapy with troglitazone and metformin in women with the polycystic ovarian syndrome, demonstrates that interventions designed to attenuate insulin resistance...

Sources of Estrogens in Women

In the circulation, estrogen binds to sex hormone binding globulin (SHBG) produced in the liver and, with less affinity, to albumin (11). Only about 2 -3 of estrogen is free. Changes in SHBG levels may influence the tissue availability of free estrogen and also free androgen because the latter also binds to SHBG. Estrogens themselves increase, whereas androgens and high insulin levels decrease SHBG levels. During the menopause, the drop of estradiol reduces SHBG levels, which in turn, results in decreased binding and an increased concentration of free androgens. Consequently, estrogens decrease to a greater extent than do androgens resulting in an increase of the androgen estrogen ratio and a relative androgen excess in postmenopausal women. Some of the signs and symptoms observed after menopause and, in particular, changes in body composition are caused by this altered balance between estrogens and androgens (12).

Relationship to Insulin Resistance and Diabetes

Although the exact mechanisms that lead to the development of PCOS are not clear it has been shown that insulin resistance and compensatory hyperinsulinemia possess the central role in the pathophysiology of the syndrome. Women with PCOS have both basal and glucose-stimulated hyperinsulinemia compared with weight-matched women and the high levels of insulin are thought to mediate the development of hyperandrogenemia, anovulation, and infertility. At the same time, insulin resistance and compensatory hyperinsulinemia are responsible for the cardiovascular risk factors. The hyperinsulin-ism correlates with the hyperandrogenism and occurs independent of obesity (180,181). The insulin resistance in at least 50 of PCOS women appears to be related to excessive serine phosphorylation of the insulin receptor (182). This abnormality is caused by a factor extrinsic to the insulin receptor, which is presumably a serine threonine kinase. Serine phosphorylation appears to modulate the activity of...

Pathophysiology

Polycystic ovary syndrome (chronic anovulation) Obesity Diabetes 1. Endogenous estrogen. One source of endogenous unopposed estrogen is chronic anovulation is associated with polycystic ovary syndrome (PCOS) and the perimenopausal period. Secretion of excessive estradiol from an ovarian tumor (eg, granulosa cell tumor) may also result in endometrial hyperplasia.

Mechanisms

Apart from the above considerations, diabetes may also affect the vascular system in women indirectly, through menstrual cycle irregularities and hypoestrogenemia. Indeed, previous epidemiological studies demonstrated that diabetic premenopausal women more frequently have menstrual irregularities, lower blood estrogen levels and higher androgen levels than nondiabetic women (118). The reasons for these menstrual abnormalities and hypoestrogenemia in women with diabetes are not well known but may be hypotha-lamic in origin related to stress or poor metabolic control or may be related to insulin resistance and hyperinsulinemia (119). Furthermore, in women with type 2 diabetes, low SHBG levels have been reported and this may contribute to relative hyperadrogenemia in these women, a condition frequently seen in women with polycystic ovary syndrome (PCOS) (120). Low SHBG levels are believed to be a marker for future CVD in women (121). Whatever the reason, the diabetes related-menstrual...

Findings

Always do a D&C to rule out endometrial cancer in women over 35. Also get hemoglobin hematocrit to make sure that the patient is not anemic from excessive blood loss. Uncommon causes of DUB are infections, endocrine disorders (thyroid, adrenal, pituitary prolactin), coagulation defects, and estrogen-producing neoplasm. Polycystic ovarian syndrome (PCOS) look for heavy woman who has hirsutism, amenorrhea, and or infertility. PCOS is the. most Likely cause of infertility in a woman, under 30 with abnormal menstruation. Multiple ovarian cysts often are seen, on ultrasound.The primary event is androgen excess. The ratio of luteinizing hormone (LH) to follicle-stimulating hormone(FSH) is greater than 2 1. Unopposed estrogen increases the risk for endometrial cancer. Treat with oral contraceptives or cyclic progesterone. If the patient desires pregnancy, use clomiphene. symptom for anorexia (amenorrhea required for a diagnosis of anorexia), especially in a ballet...

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