How I Healed my Varicose Veins
Treatment for varicose veins is aimed at improving blood flow, reducing injury, and reducing venous pressure. Pharmacologic treatment is not indicated for varicose veins. To give support and promote venous return, physicians recommend wearing elastic stockings. If the varicosities are moderately severe, the physician may recommend antiem-bolism stockings or elastic bandages or, in severe cases, custom-fitted heavy-weight stockings with graduated pressure. When obesity is a factor, the patient is placed on a weight loss regimen. Experts also recommend that the patient stop smoking to prevent vasoconstriction of the vessels. A nonsurgical treatment is the use of sclerotherapy for varicose and spider veins. Sclerother-apy is palliative, not curative, and is often done for cosmetic reasons after surgical intervention. A sclerosing agent, such as sodium tetradecyl sulfate (Sotradecol), hypertonic saline, aethoxysclerol, or hyperosmolar salt-sugar solution, is injected into the...
The anti-inflammatory and analgesic effects of TD GTN was studied in 21 patients with mild to moderate leg varicose veins who underwent vein sclerotherapy in both legs (139). The vein in one leg was treated every 8 h with GTN and compared with a placebo ointment applied to the vein of the other leg. Inflammation signs were observed in all cases 15 min after first application. Intensity of inflammation signs were 26 in GTN-treated veins and 61.5 in placebo-treated veins. One hour later only 63 of cases in the GTN group, but all cases in the placebo group, showed signs of thrombophlebitis. All veins in the GTN group were free of signs of thrombophlebitis in fewer than 48 h, whereas, of the placebo group, 45 required more than 48 h.
The role of platelets in the recruitment of monocytes and neutrophils to sites of vascular injury has long been recognized (Palabrica et al., 1992). The appearance of circulating LPAs is a dynamic process involving initial formation, vascular adhesion, potential sequestration to elements of the reticuloendothelial system, leukocyte activation, and LPA disaggregation via granulocyte proteinases (Gardiner et al., 2001). Circulating LPAs are increased in stable coronary artery disease (Furman et al., 1998), unstable angina (Ott et al., 1996), acute myocardial infarction (Furman et al., 2001 Michel-son et al., 2001), chronic venous insufficiency (Powell et al., 1999), and during cardiopulmonary bypass (Rinder et al., 1992). Circulating LPAs are also increased after coronary angioplasty, with a greater magnitude in patients experiencing late clinical events (Mickel-son et al., 1996).
On examination, the lower leg and ankle and foot may be edematous, there may be prominent varicose veins present. Leakage of blood into the skin may cause deposition of hemosiderin. The ulcers may heal spontaneously or may become chronic and indolent. The ulcers may be complicated by infection, bleeding or by eczema. Varicose eczema, surrounding the ulcer, is common, and in many patients, it may be due to allergic contact hypersensitivity to medicaments, such as neomycin clioquinal (Vioform), lanolin, or ethylene diamine, used in treating the leg ulcer or eczema. Rarely, in long-standing ulcers, malignant change with the development of squamous cell carcinoma may occur, and patients with long-standing ulcers may become anemic. It is important to try and improve venous return (drainage) by
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