Cure For Vitiligo Found

Natural Vitiligo Treatment System

In this ebook, Michael Dawson identifies the root cause of the disease and offers solution that is more than just surface effects. He says that following a holistic approach to proper diet and making lifestyle changes, the disease can truly be defeated. One of the important claims of this treatment is that it is totally natural and has no side effects like other treatments. The treatment aims to provide a natural and effective solution that can bring an end to this disease. Natural Vitiligo Treatment System offers a cheap and innovative solution that is suitable for all individuals. The steps are easy to follow and the natural substances that are prescribed are less expensive than what other conventional treatments for Vitiligo require. In addition, it also improves the body immunity and the general health of a person. This ebook is a mine of information on the treatment for Vitiligo and allows users to nourish their body with the right nutrients.

Natural Vitiligo Treatment System Overview


4.6 stars out of 11 votes

Contents: 60+ Page Ebook
Author: Michael Dawson

Download Now

Vitiligo Miracle

Vitiligo Miracle is an ebook that was written by David Paltrow. The author of this ebook is a nutritionist, a medical researcher who has twelve years experience, and works as a health consultant. This program focuses on internal factors that are the root causes of Vitiligo with a holistic, multi-dimensional approach and fixes it permanently. The biggest problem with this diet is that persons may have trouble developing vitiligo management habits, but because of the nature of the condition most persons are heavily motivated to do so.

Vitiligo Miracle Overview

Contents: EBook
Author: David Paltrow
Official Website:
Price: $37.00

Fast Vitiligo Cure

In this 70-page digital e-book, you will find the only step-by-step, proven natural Vitiligo cure method in existence and be able to cure any Vitiligo skin disorder in less than 30 days. The proven 7 step formula that has helped thousands of children and adults. cure Vitiligo in 30 days or less. The top 12 home remedies, with step-by-step instructions, that will. help get rid of patches of discolored skin and restore skin immediately. Discover Everything you need to know about Vitiligo, Exactly what causes it. and more importantly, strategies on how to get rid of it. The top 5 bathing procedures to relieve any itchiness or discomfort from the. Vitiligo patches immediately, as well as help heal the patches and skin quickly. The top 10 best foods to boost your immune system and help your body. function properly in producing healthy skin cells quickly. The top 3 worst foods to avoid and should Never eat when suffering from Vitiligo. How to prevent any ugly scars or marks from showing up. and how to get. rid of any Vitiligo scars by following these simple treatments. Secret: Discover one of the most powerful natural supplements that when used will. help purify the blood, boost the immune system and help it function properly to have. natural, healthy skin. and is 100% safe and effective! Quickly reverse most Vitiligo symptoms with this effective superfood. that you won't find anywhere else on the internet! The best full-spectrum multi-vitamin and mineral supplement that you should be. taking every day, and will immediately help speed up recovery of Vitiligo. (hint. most people aren't giving their bodies the proper vitamins, minerals, and nutrients that the body needs to win this battle). The biggest mistake that every Vitiligo sufferer makes, which is. preventing the body from being able to get rid of the skin disorder quickly. by weakening the immune system, thus making you suffer More and. Longer than necessary. How to avoid scratching the itchy Vitiligo patches and specific strategies to fight the. itchy temptation, so you no longer have to worry about the possibility of scarring. On page 41, this technique has been voted he best since it fixes the. root cause of Vitiligo and eliminates the patches literally within weeks. A powerful home-made smoothie recipe that tastes delicious and gives your body. Exactly what it needs to fight off this skin disorder and provide a huge boost of energy. Warning: On page 59, you will discover a very common household. ingredient that has the ability to aggravate your Vitiligo and prevent. your body from healing quickly. If you do not get rid of this and stop. consuming it, you are in for a life-long battle with Vitiligo. The top 4 most powerful supplements that no doctor will ever tell you about, that will. help strengthen the immune system and speed up recovery time.

Fast Vitiligo Cure Overview

Contents: 70-page Digital E-book
Author: William Oliver
Official Website:


Gauthier Y, Cario Andre M, Taieb A. A critical appraisal of vitiligo etiologic theories. Is melanocyte loss a melanocytorrhagy Pigment Cell Res 2003 16(4) 322-332. 3. Hann SK, Park YK, Lee KG, Choi EH, Im S. Epidermal changes in active vitiligo. J Dermatol 1992 19 217-222.

Indicated Supporting Diagnostic Data

Dermatology Biopsy Tools

A 25-year-old woman requests evaluation for symmetric white macules and patches on her neck and upper torso. She has recently read about vitiligo on the internet and is terrified of permanent disfigurement. Examination reveals oval thumbprint-size white macules and larger confluent patches with smooth margins. Gentle scraping of a lesion raises a loose white scale. A 25-year-old woman requests evaluation for symmetric white macules and patches on her neck and upper torso. She has recently read about vitiligo on the Internet and is terrified of permanent disfigurement. Examination reveals oval thumbprint-size white macules and larger confluent patches with smooth margins. Gentle scraping of a lesion raises a loose white scale.

Subacute Cutaneous Lupus Erythematosus

Subacute Cutaneous Lupus

Initially, SCLE lesions present with erythematous macules and papules that evolve into scaly papulosquamous or annular polycyclic plaques (Sontheimer et al. 1981). Approximately 50 of patients have predominantly papulosquamous or psoriasiform lesions (Fig. 6.4), and the other half have the annular polycyclic type (Fig. 6.5) a few patients may develop both forms of lesions (Sontheimer 1985a, Sontheimer et al. 1979). However, some groups have observed a predominance of the papulosquamous lesions, whereas others have noted an abundance of the annular polycyclic type (Callen and Klein 1988, Chlebus et al. 1998, Cohen and Crosby 1994, David-Bajar 1993, Fabbri et al. 1990, Herrero et al. 1988, Molad et al. 1987). One recent study found that 42 of the patients with SCLE studied exhibit the annular polycyclic form, 39 had the papulosquamous form, and 16 showed both manifestations (Parodi et al. 2000). Generally, lesions of this subtype heal without scarring but can leave long-lasting and...

Step 2 Assess the history

Measurements of height and weight, signs of other illnesses, and evidence of cachexia should be assessed. The skin, breasts, and genital tissues should be evaluated for estrogen deficiency. The breasts should be palpated, including an attempt to express galactorrhea. The skin should be examined for hirsutism, acne, striae, acanthosis nigricans, vitiligo, thickness or thinness, and easy bruisability.

Lupus Erythematosus and Sjgrens Syndrome

Anti-La SSB occurs in approximately 15 of patients, and most have SS and may be diagnosed as having SS-SLE overlap syndromes. Anti-Ro SSA (without anti-La SSB) occurs in approximately 30 of patients with SLE without dry eyes or mouth. SS can precede LE by years, but LE can precede the appearance of SS also. Anti-Ro SSA antibodies etc found in polymyositis, scleroderma, and primary biliary cirrhosis, and association with SS is invariably (Venables 1988). The skin changes associated with SS include dry eyes, dry mouth, and dry genitals as major clinical symptoms. Other clinical symptoms include annular erythema, Sweet's syndrome-like lesions, vitiligo-like changes, sarcoidosis, and LP-like and amyloidosis nodularis-like symptoms (Ueki 1994, personal communication). Annular erythema is one of the prominent clinical features of both SS and SLE. Katayama et al. (Katayama et al. 1991) subdivided annular erythema into three types. Type I is an isolated annular erythema with an elevated and...

Morphologic and Physiologic Skin Differences in Dark Skin

Dark skin demonstrates significantly greater intrinsic photoprotection because of the increased content of epidermal melanin. Clinical photodamage, actinic keratoses, rhytides, and skin malignancies are less common problems in deeply pigmented skin. However, darker skin types are frequently plagued with dyschromias because of the labile responses of cutaneous melanocytes 7 . In a survey of 2000 black patients seeking dermatologic care in a private practice in Washington, DC, the third most commonly cited skin disorders following acne and eczema was pigmentary problems other than vitiligo 8 . Of these patients, the majority had a diagnosis of post-inflammatory hyper-pigmentation, followed in frequency by melas-ma. In a survey of 100 women of color assessing issues of cosmetic concerns for darker skin types, the most commonly cited problems were dark spots or blotchy skin, texturally rough skin, and increased sensitivity to topical products 9 . Patients surveyed also complained of oily...

Halo Melanocytic Nevus

Halo nevi are common nevi exhibiting a peripheral vitiligo-like annulus or halo of hypopigmentation or depigmentation surrounding a central nevus (Fig. 4A). Histologically halo nevi demonstrate a dense lymphocytic infiltrate associated with the central nevus and a peripheral zone of hypo- to depigmentation of the epidermis, corresponding to the clinical halo (Fig. 4B). Halo melanocytic nevi may exhibit both clinically and histologically atypical features and thus raise concern for melanoma (Tables 4 and 7).

Safety Concerns Regarding the Use of Recombinant Viruses

Vaccinia virus (VV) and adenovirus have a very broad host range, infecting virtually all mammalian cells, including human. Everyone born before the early to mid-1970s has been vaccinated with the smallpox vaccine, which provides a long-lasting protection against systemic viremia upon VV infection. Most people are continually exposed to adenovirus, which is present in the upper respiratory tract of a large portion of healthy individuals where it ordinarily does not cause pathology. However, it is important to realize that, even in individuals who have been repeatedly boosted, a limited infection does occur upon dermal scarification with VV, as evidenced by a localized pustule and tender draining lymph nodes. Evidence is accumulating that mucosal immunity to vaccinia may be weak, so protection from infection through nose, mouth, and eyes may be particularly poor. In addition, wild-type adenoviruses cause significant numbers of respiratory tract infections and even deaths, especially in...

Old burntout Lesions of Discoid Lupus Erythematosus

Lesions of vitiligo may closely resemble burnt-out DLE, with regular and only mildly altered surface texture because of its round shape and circular outlines. Lesions must be carefully examined for minimal signs of scarring. At the clinical overview, vitiligo is characterized by its larger lesions and its predilection for periori-ficial location.

Psoriasis and Lupus Erythematosus

The control of SLE often requires systemic administration of steroids, especially for renal and central nervous system involvement. A rebound flare of psoriasis is always possible on withdrawal of steroid therapy. Administration of antimetabolites used as steroid-sparing agents may prevent this rebound flare and improve psoriasis. Phototherapy is contraindicated in patients with cutaneous LE. On the contrary, pso-ralen-UVA exposure is indicated in psoriatic individuals and in those with severe photosensitive psoriasis. Screening for ANAs, including anti-Ro SSA and anti-La SSB antibodies, is necessary before treating any photosensitive patient with UV light. Psoriasis could coexist with other photosensitive disorders, such as vitiligo, por-phyria, drug-induced photodermatitis, polymorphous light eruption, chronic actinic dermatitis, solar urticaria, actinic prurigo, and the so-called fair skin type.

Tumor Protection Using Gmcsftransduced Wholecell Vaccine B16gmcsf

It is difficult to induce reliable protection against B16 challenge by vaccination with irradiated B16, even when admixed with Corynebacterium parvum. However, robust protection can be obtained by vaccinating with B16 that is retrovirally transduced to secrete high levels of GM-CSF (Dranoff et al., 1993). Although B16.GM-CSF will still grow upon injection, vaccination with irradiated cells will induce a T cell-dependent protection against wild-type B16. It is unknown what antigens are targets of this immune protection, and the involvement of eosinophils and macrophages has been implicated (Hung et al., 1998). The following protocol describes the use of B16.GM-CSF for protection against B16 challenge in the authors ' laboratory. Recent results suggest it may also be possible to impact on growth of established tumors by vaccinations with irradiated B16.GM-CSF, especially in conjunction with anti-CTLA-4 antibody (van Elsas et al., 1999). The addition of this antibody, which presumably...

B16 as a Mouse Model for Human Melanoma

Since MDA are expressed by most melanoma cells, they are an attractive target for melanoma vaccines. However, from an immunological perspective, MDA are self proteins, to which central and or peripheral tolerance may exist, potentially hampering the induction of powerful, therapeutic anti-melanoma immune responses. Yet clinical observations suggest that some degree of autoreactivity can be induced and may even contribute to prolonged survival of patients. In a prospective study of patients receiving IL-2, vitiligo was seen in 20 of melanoma patients that had objective responses. None of the nonresponding patients developed vitiligo, nor did any of the more than 100 patients receiving IL-2 for the treatment of kidney cancer (Rosenberg and White, 1996). This unit will detail protocols for in vivo models of subcutaneous growth and pulmonary metastases of B16 melanoma (see Basic Protocols 1 and 2). Therapeutic approaches include the use of B16.GM-CSF (see Basic Protocol 3) and rVVmTRP-1...

Classification of Diabetes Mellitus and the Metabolic Syndrome

Immune-mediated diabetes, previously referred to as insulin-dependent diabetes, type I diabetes, and juvenile-onset diabetes, accounts for 5-10 of all cases of diabetes. Immune-mediated diabetes typically develops in childhood and adolescence, but has a variable age of onset ranging from infancy to the eighth and ninth decades of life. Abnormalities in glucose homeostasis result from severe insulin deficiency due to cell-mediated autoimmune inflammation (insulitis) and subsequent destruction of the b-cells of the pancreas. In T1DM, daily exogenous insulin administration is a life-sustaining intervention, and the absence of insulin can result in a state of acute metabolic decompensation known as diabetic ketoacidosis (DKA). Individuals with T1DM are at increased risk for other autoimmune disorders such as Graves' disease, Hashimoto's thyroiditis, Addison's disease, vitiligo, celiac sprue, autoimmune hepatitis, myasthenia gravis, and pernicious anemia.

List of Contributors

(e-mail pegrimesmd Vitiligo and Pigmentation Institute of Southern California and Division of Dermatology, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, California, 5670 Wilshire Blvd., Suite 650, Los Angeles, CA 90036, USA (e-mail tsoriano Vitiligo and Pigmentation Institute of Southern California and Division of Dermatology, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, California


Glycolic acid has been recognized as an important adjunctive therapy in a variety of conditions including photodamage, acne, rosacea, striae albae pseudofolliculitis barbae, hyper-pigmentation disorders, actinic keratoses, fine wrinkles, lentigines, melasma and seborrheic keratoses 5 . Moreover, it can reduce UV-in-duced skin tumor development and it has been proposed as a therapeutic modality against skin exfoliative conditions such as ichthyosis, xeroderma and psoriasis. In post-menopausal women a cream containing 0.01 estradiol and 15 glycolic acid, applied to one side of the face for 6 months, induces a significant improvement in reversing markers (rete peg pattern, epidermal thickness) of skin aging 6 .

Specific History

A careful medication history is essential, and any medication reported to cause an LP-like eruption should be discontinued. The features of these drug eruptions are sometimes strikingly similar to the idiopathic disease both clinically and microscopically, and offending drugs will be uncovered only by careful history. The list of medications that cause these reactions continues to grow and any agent should be suspect. These drug-induced eruptions are slow to clear and it is not unusual for improvement to take 2 or 3 months. LP has also been associated with an increased incidence of autoimmune diseases (Sjogren's syndrome, sicca syndrome, alopecia areata, vitiligo, ulcerative colitis, myasthenia gravis, and diabetes mellitus), chronic dermatophyte infections, and chronic liver disease (primary biliary cirrhosis, alcoholic cirrhosis, chronic active hepatitis B and C). The presence of a fungal infection is usually significant the authors have seen many cases of LP that clear and...

Lichen Planus

Lichen planus may be associated with other autoimmune diseases, including vitiligo and myasthenia gravis. The etiology is unknown but the deposition of IgM at the dermoepidermal junction, coupled with a dense inflammation in the upper dermis, suggest an autoimmune process. Lichen planus-like rashes may be precipitated by various drugs, including thiazide diuretics, gold, tetracyclines, and para-aminosalicylic acid (PAS). The disease is usually self-limiting, but topical cortico-steroids may be helpful.

Dating of Contusions

The method most commonly employed in dating contusions is based on the changes in color a contusion undergoes as it heals. The depth of a contusion and skin pigmentation may affect the appearance and detection of the colors. Yellow coloration is visible sooner in superficial bruises than in deep bruises dark pigmentation may conceal a bruise. The depth and location of the bruise can influence its time of appearance, with superficial bruising and bruises of the eyelids (with their loose soft tissue) appearing immediately, and deep bruising not appearing for days.


Ness, drowsiness, fatigue, hallucinations, insomnia, lethargy, mental changes, memory loss, strange dreams. GI Diarrhea, ischemic colitis, nausea, mesenteric arterial thrombosis, vomiting. Hematologic Agranulocytosis, thrombocytopenia. Allergic Fever, sore throat, respiratory distress, rash, pharyngitis, laryngos-pasm, anaphylaxis. Skin Pruritus, rash, increased skin pigmentation, sweating, dry skin, alopecia, skin irritation, psoriasis. Ophthalmic Dry, burning eyes. GU Dysuria, impotence, nocturia. Other Hypoglycemia or hyperglycemia. Respiratory Bronchospasm, dyspnea, wheezing. Drug Interactions See also Drug Interactions for Beta-Adrenergic Blocking Agents and Antihypertensive Agents.


Among the several cases reported is one with hydralazine-induced SLE presenting as pyoderma gangrenosum-like ulcers (Peterson 1984). There are occasional studies about the association of LE with hereditary angioedema. All subtypes of LE have been reported to be coexistent with hereditary angioedema (see Gudat and Bork 1999). Duhra et al. (Duhra et al. 1990) reported in 1990 a female patient who developed DLE 6 years after the onset of hereditary angioedema. They tried to explain the coexistence of both diseases by C1 inhibitor deficiency, which can induce DLE. Duhra et al. believed that this association is rare, but its recognition is important because both diseases respond to danazol therapy. A single case showing an association between DLE and pseudoainhum has been reported (Sharma et al. 1998). Pseudoain-hum is a rare dermatologic complication presenting as a constricting band around the digits. The reported patient was a 32-year-old male with 10 years duration of DLE....

Causes of anemia

Vitamin B)2 deficiency (macrocytic) most commonly due to pernicious anemia (antiparietal cell antibodies). Remember the physiology ofBJ2 absorption and the association with vitiligo and hypothyroidism. Other causes include gastrectomy, terminal ileum resection, diet (strict vegan), chronic pancreatitis, and Diphyllobothiium latum (fish tapeworm) infection. Peripheral smear looks the same as in folate deficiency (macrocytes, hypersegmented neutrophils), but the patient has neurologic deficiencies (loss of sensation or position sense, paresthesias, ataxia, spasticity, hypcrreflexia, positive Babinskt sign, de mentia). Look for low serum B13 , achlorhydria (no stomach add secretion, elevated stomach pH), and antibodies to parietal cells. A Schilling test usually determines the etiology. Usual replacement route is intramuscular, because most patients cannot absorb Bl2.

Antileprotic Drugs

Clofazimine is a dye with bactericidal activity against M. leprae and anti-inflammatory properties. It is given orally, but is incompletely absorbed. Because of its high lipophilicity, it accumulates in adipose and other tissues and leaves the body only rather slowly (t1 2 70 d). Red-brown skin pigmentation is an unwanted effect, particularly in fair-skinned patients.

Phenol Peels

Skin Depigmentation Clinically and histologically, patients undergoing phenol chemical peeling may exhibit a bleaching effect. After phenol peeling, melanocytes reorganize themselves along the basement membrane. Although still present, these melanocytes lose their ability to produce melanin. Clinically, this results in a bleached appearance. This may be desired and beneficial for those patients seeking treatment of certain pigmentary problems. However, in patients undergoing regional facial peeling, this bleaching effect may become noticeable and problematic. It is often most noticeable in the jaw-neck region, where untreated skin in the neck appears more obvious as it abuts the newly rejuvenated cheek or periorbital skin.

Intermezzo Example

In the city of Antofagasta, Chile, the water supply contained high concentrations of arsenic during the period between 1958 and 1970. In this desert area, the water has to be brought from the Andes, where arsenic is found in naturally high concentrations, over a distance of 300 km. Copper mining was another probable source of arsenic contamination. In the early 1960s, a large number of citizens (children in particular) were found to have an abnormal skin pigmentation. Clinical investigations revealed that this pigmentation was